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Article summary:

1. Abrogation of HnRNP L enhances anti-PD-1 therapy efficacy in castration-resistant prostate cancer.

2. This is achieved by diminishing PD-L1 and promoting CD8+ T cell-mediated ferroptosis.

3. The study suggests that targeting HnRNP L may be a potential strategy to improve the response to anti-PD-1 therapy in prostate cancer patients.

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