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1. BPS exposure during pregnancy can lead to hepatic lipid deposition in male mice offspring: The study found that prenatal exposure to bisphenol S (BPS), a chemical commonly used in various products, can result in the accumulation of lipids in the liver of male mice offspring. This suggests that BPS exposure during pregnancy may have adverse effects on lipid metabolism in offspring.

2. Adipose-derived exosomal miRNAs play a role in regulating hepatic lipid metabolism: The researchers identified key adipose-derived exosomal microRNAs (miRNAs) that are involved in regulating hepatic lipid deposition. These miRNAs are transported by exosomes, which are extracellular vesicles, and can influence gene expression in the liver. Understanding this mechanism could provide insights into the toxic effects of prenatal BPS exposure on offspring.

3. Prenatal BPS exposure affects body weight and lipid metabolism: The study found that male mice offspring with prenatal BPS exposure had increased body weight compared to the control group. Additionally, BPS exposure was associated with disruptions in lipid metabolism, which is closely related to conditions such as obesity and non-alcoholic fatty liver disease (NAFLD). These findings highlight the potential health risks of prenatal BPS exposure and its impact on metabolic health.

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