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Article summary:

1. Lumpy skin disease virus (LSDV) activates the unfolded protein response (UPR): The article presents emerging evidence that LSDV, a poxvirus, triggers the UPR in infected cells. Specifically, it activates two UPR signaling pathways, PERK and IRE1.

2. Activation of PERK and IRE1 promotes self-replication of LSDV: The study suggests that by activating PERK and IRE1, LSDV is able to maintain its self-replication within host cells. This finding sheds light on the mechanisms underlying the replication and persistence of poxviruses.

3. Potential implications for understanding other diseases: The activation of UPR signaling pathways by LSDV may have broader implications beyond lumpy skin disease. It could provide insights into the pathogenesis of other viral infections and potentially lead to the development of novel antiviral strategies targeting the UPR.

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