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Article summary:

1. Neuroinflammation can lead to synapse dysfunction and cognitive decline.

2. The transcriptional repressor REST is activated by neuroinflammatory stimuli and upregulation of REST can lead to synaptic downscaling, specifically for excitatory synapses.

3. IL-1β is a potent cytokine that can cause a prompt increase in REST transcription and translation in neurons, leading to the observed synaptic dysfunctions under neuroinflammatory conditions.

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