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Cellular senescence promotes cancer metastasis by enhancing soluble E-cadherin production - PubMed
Source: pubmed.ncbi.nlm.nih.gov
Article summary:
1. Cellular senescence promotes cancer metastasis in a non-cell-autonomous manner.
2. An antibody array screening identified soluble E-cadherin (seCad) as a potential mediator of the senescence-induced melanoma metastasis.
3. Serum seCad is associated with distant metastasis, suggesting that it may be a potential diagnostic marker and therapeutic target for metastatic lung cancer.
Article analysis:
The article is generally reliable and trustworthy, as it provides evidence to support its claims and presents both sides of the argument equally. The authors have conducted extensive research to identify seCad as a potential mediator of the senescence-induced melanoma metastasis, including an antibody array screening, gene expression profiling, and analysis of sera from patients. Furthermore, they provide evidence that serum seCad is associated with distant metastasis, suggesting that it may be a potential diagnostic marker and therapeutic target for metastatic lung cancer.
The article does not appear to contain any promotional content or partiality towards one side of the argument over another. It also does not appear to contain any unsupported claims or missing points of consideration; all claims are backed up by evidence from the research conducted by the authors. Additionally, possible risks are noted throughout the article, such as the risk of developing distant metastases in patients with melanoma.
In conclusion, this article is reliable and trustworthy due to its thorough research methods and balanced presentation of both sides of the argument.